According to a new study published in theEuropean Respiratory Journal, the vapor emitted by electronic cigarettes increases the sensitivity to lung infections just like the smoke of traditional cigarettes. It took a short time before several experts denounced the methodology of this study which once again comes to harm the vaping.
A study of theQueen Mary University of London (Great Britain) reveals that vaping makes the airways as vulnerable to bacteria that adhere to airway cells as smoke from traditional cigarettes or exhausts, increasing the risk of respiratory infection in people the most vulnerable.
The researchers examined the effects of vaping on a cell-based molecule that lines the airways called platelet factor receptor (PAFR), which helps the bacteria that cause pneumonia stick to the nose, throat and lungs. Previous studies have shown that levels of PAFR increase in response to smoking, passive smoking and exhaust fumes.
To see if the effect was the same with e-cigarettes, they studied the cells that lined the nose of 17 e-cigarette users one hour after the vaping. Of these, 10 were regular users of nicotine-containing electronic cigarettes, 1 used nicotine-free e-cigarettes and 6 were not regular vapers. They found that the levels of LCP had tripled from normal levels.
A SATISFACTORY METHODOLOGY ACCORDING TO CERTAIN EXPERTS
Following this study, researchers suggest that people at high risk of pneumonia who would like to quit smoking opt instead for nicotine patches or gums as an aid to smoking cessation. While the use of electronic cigarettes is clearly not risk-free, yet they remain at least 95% less harmful than fuel cigarettes according to Public Health England.
Besides the teacher Peter Hajek, director of the Tobacco Dependence Research Unit, QMUL, said following this study:
« It is a shame that the study did not compare its cellular effects with those of smoking. The effects of the aerosol of the e-cigarette were compared to the effects of clean air but it was more important to compare it with smoking. No matter the comparator, we do not really know if there is something alarming in these results. The most relevant part of the article concerns cells taken from people whodo not smoke or vaping. There was no difference in PAFR levels between vapers and non-vapers in the main samples ! The study only noted a transient acute effect after vaping. How can this translate to health? It is not clear. Data from people, as opposed to cells and animals exposed in very different ways, show no evidence of increased susceptibility to vaping infections following e-cigarette use. results point in the opposite direction, previous work suggests that smokers who switched from smoking to smoking did not report an increase, but rather a significant decrease in respiratory infections. ".
« The results of this study with lab-grown cells and mice suggest that vaping could make cells lining the airways more sticky and thus more susceptible to bacterial colonization, but this remains indirect evidence that vaping may increase risk of lung infection in humans. Although it is possible that vaping may increase the risk of pneumonia, the risk remains clearly lower than with smoking. We need further research to determine whether vaping in comparison to smoking is likely to increase the risk of pneumonia. This study should not be used to encourage smokers not to use the e-cigarette. To date, there is evidence that e-cigarettes are much less harmful than smoking. »